More recently, some groups have found decreased IOP, 2others have found increased IOP, 3 4normal IOP, 5 6and even a reduction in IOP that occurred within hours of ascent and recovered during acclimatization. In 1918 Wilmer and Berens 1measured IOP in 14 aviators in a hypobaric chamber but found no significant changes. Intraocular pressure (IOP) at high altitude has been the subject of controversy for many years. Changes in IOP at altitude are not predictive of symptoms of acute mountain sickness (AMS) or development of high-altitude retinopathy (HAR). IOP returned to baseline levels and possibly lower with prolonged exposure to altitude. This finding may be partially explained by the change in CCT. Acute exposure to altitude caused a statistically significant but clinically insignificant increase in IOP. IOP at baseline, change in IOP from baseline, and IOP at altitude did not predict symptoms of acute mountain sickness (AMS) or development of high-altitude retinopathy (HAR).Ĭonclusions. IOP increased significantly from baseline after acute exposure to altitude before returning to baseline with time. Pre- and postexpedition CCT and postexpedition IOP readings at sea-level were also measured. IOP and CCT were measured with a hand-held tonometer and ultrasound pachymetry on the first, third, and seventh days at 5200 m. They all arrived in La Paz, Bolivia (altitude, 3700 m), where they spent 4 days before being driven more than 2 hours to the Cosmic Physics Laboratory at Chacaltaya (5200 m) where they stayed for 7 days. The Apex 2 medical research expedition provided the opportunity to measure intraocular pressure (IOP) and central corneal thickness (CCT) in 76 healthy lowlanders. To investigate changes of intraocular pressure on ascent to high altitude.
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